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The peptide upregulates MMP-2 expression in dermal fibroblasts by 85%, facilitating breakdown of damaged collagen and enabling cellular migration through dense matrix environments, while simultaneously enhancing production of tissue inhibitors of metalloproteinases (TIMPs) to prevent excessive degradation
Proper technique maintains peptide integrity and ensures accurate dosing
GHK-Cu, delivered simultaneously to those same fibroblasts, amplifies the collagen signal while suppressing the excess inflammatory response
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