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This far exceeds the affinity of many endogenous copper-chelating agents, and it matters because that tight coordination is what allows GHK-Cu to donate copper to cuproenzymes and initiate downstream gene expression cascades

Interest in such applications is largely driven by their proposed mechanisms of action, including enhancement of tissue repair, modulation of inflammation, and cytoprotective effects observed in preclinical studies

[10] Long-term use of proton pump inhibitors (PPIs) and H2-receptor antagonists reduces gastric acid, potentially impairing the release of B12 from dietary proteins

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