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After 48 h incubation at 27C, the cells were collected by centrifugation and stored at 80C until use
CETP mice injected with either vehicle, a GIPR agonist (GIPFA-085), a GLP1R agonist (GLP-140) or both agonists presented similar results ( and proatherogenic peptides and systemic inflammation markers ICAM-1 and VCAM-1 involved in monocytes/macrophages adhesion to endothelium
The logic isn't "more peptides = better." It's that these two peptides address different bottlenecks in the healing process
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