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Therefore, disturbing the autophagylysosome function is efficient for delaying the occurrence and development of tumors, and it is gradually becoming a promising target for tumor treatment
For the injectable formulation at 2.4 mg, the reported rate was lower but still present: one of 407 patients reported dysesthesia
Finally, we establish that this pathway is conserved in human ILC2s and can be therapeutically manipulated using clinically relevant NRF2-modulating agents
10.1016/j.bcp.2021.114786 84
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