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Switching back to mammalian models (hamster, rats, pigs), researchers found that GLP-1 could stimulate insulin secretion and inhibit glucagon release, which could vastly benefit diabetic patients

The identification of these alternative immune checkpoints highlights the need for multi-targeted immunotherapeutic approaches that nanomedicine platforms are uniquely positioned to deliver ( Figure 1 )

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3 Because postprandial hyperglycemia affects A1C to a greater degree than fasting hyperglycemia, the closer a patient is to the A1C goal, GLP-1 receptor agonists provide a viable treatment option to target postprandial glucose excursions due to their glucose-dependent effects on insulin secretion